Wen Zeng | Molecular and Cellular Neuroscience | Best Researcher Award

Prof. Wen Zeng | Molecular and Cellular Neuroscience | Best Researcher Award

Prof. Wen Zeng | Army Medical University | China

Wen Zeng is a Professor and Chair of the Department of Cell Biology at the College of Basic Medical Sciences, Army Medical University, where he leads a multidisciplinary team dedicated to advancing the understanding and treatment of neurovascular dysfunction, a critical pathological factor underlying many human diseases. His research centers on elucidating the mechanisms governing neurovascular interactions and translating these insights into innovative cell-based therapeutics, particularly through the use of stem and progenitor cells to regenerate damaged neurovascular networks and promote structural and functional recovery in tissues such as the heart, brain, and bone. Professor Zeng has completed and is currently leading numerous competitive research projects and has built a strong record of scholarly productivity, with dozens of publications in high-impact SCI and Scopus-indexed journals and an H-index of 68 in the Web of Science Core Collection, reflecting the broad influence of his work. He is also an active innovator, holding multiple patents that support the clinical translation of his discoveries, and has engaged in consultancy and industry-oriented projects to bridge basic research with real-world applications. Through international collaboration, notably with Dr. Mengqiu Xing at the University of Manitoba, his team has developed a microchannel hydrogel suture capable of bidirectional signal communication, on-demand drug delivery, and cardiac repair, highlighting his commitment to interdisciplinary research and translational medicine aimed at improving outcomes in complex neurovascular and cardiovascular disorders.

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Featured Publications

Application progress of bio-manufacturing technology in kidney organoids

Artificial biomarker-based feedback-regulated personalized precise thrombolysis with lower hemorrhagic risk

Engineered exosomes reprogram Gli1+ cells in vivo to prevent calcification of vascular grafts and autologous pathological vessels

Programmable dual responsive system reconstructing nerve interaction with small-diameter tissue-engineered vascular grafts and inhibiting intimal hyperplasia in diabetes

Programming of regulatory T cells in situ for nerve regeneration and long-term patency of vascular grafts